Endocarditis pada Hewan

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Endocarditis pada Hewan

cardiovascular disease is a general term used to describe diseases of the heart and blood medulla. This disease is caused by coronary arteries that deliver blood to the heart muscle, causing heart failure (coronary heart failure). If circulation is reduced, this can lead to progressive myocardial infarction (delayed heart muscle damage).

Circulatory heart, that is, blood enters the upper coronary artery from the inferior vena cava to the right atrium , then through the tricuspid valve to the right ventricle, and then to the lungs, through the pulmonary valve, through the pulmonary valve. . Through the pulmonary artery, the atria can penetrate the tricuspid valve. Through the pulmonary vein, into the atrial cavity, then the mitral or bicuspid valve into the lumen of the ventricle, and then through the aortic valve into the aorta and diarrhea.

The heart layer consists of 4 layers, namely the pericardium, epicardium, myocardium and endocardium in sequence from the outer layer to the outer layer. Endocarditis is called endocarditis when the endocardium is damaged.

Endocarditis was first discovered by Rivera in 1946

gambler. Heart inflammation.
(No: lifelearn.inc)

Most common endocarditis: SBE: subacute bacterial endocarditis, ABE: bacterial endocarditis, NVE: endocarditis, PVE: prosthetic endocarditis, NBTE: thrombotic non-bacterial endocarditis. The most commonly used terms are SBE and ABE.

There are two kinds of bacterial endocarditis (EB) namely: The first is acute EB, if the incubation period is less than four weeks. Subacute bacterial endocarditis/crohn's, especially more than four weeks, may involve endocarditis Bacterilis lanta or special tape.

Of all the disorders of the jungtungs that are often co-occurring, poppy EB is the jujube jungtung disease according to the leaders of the week: multiple sclerosis, hypertension, and rheumatic heart. EB is usually caused by congenital diseases, such as congenital atrial septal defect (ASD), ventricular septal defect (VSD), tetralogy of Fallot (TF), patent ductus arteriosus (PDA), and stenosis. Bacterial endocarditis can occur in rheumatic diseases (RHD).

2.1 Etiology
Many species of bacteri ve fungi have been reported for intermittent chlamydial endocarditis; Bacteria Nameon Demekian, Kessel's sentence From Janis with us is Jean Majority Kilimada. Benifaya microorganisms differ from endocarditis, in part as a site of entry for bacteria (Portal).

From mouth, mouth, respiratory organs, streptococci, staphylococci, HACEK organisms ( Haemophilus , Actinobacillus, Cardiac, Echinella menella ) etc. 

Streptococcus bacteria in two-zhenis: hemolytic streptococci (beta-streptococci), middle-aged hemolytic streptococci, non-hemolytic streptococci, including: green streptococci (alpha streptococci), hemolytic streptococci.

Sites of Streptococcus viridans include the oropharynx, nasopharynx, and dental carcinoma. Occasionally there is mesilic infection (mixed infection) Acute bacterial endocarditis, pneumonia, pneumonia, beta-hemolytic streptococci and Staphylococcus aureus.

Endocarditis in young people is caused by the use of injection drugs, especially in tricuspid cats, heart infections, usually by Streptococcus aureus , and methicillin-resistant young people.

2.2 tilt
There are two risk factors for endocarditis:
1. Yantong's Reason
Cardiac rheumatic disease is a factor. Preparation for endocarditis Oama Tiradinan. Congenital abnormalities Make up 6-20% susceptible to endocarditis. Congenital endocarditis: ventricular septal defect, aortic arch, coronary insufficiency, patent ductus arteriosus, tetralogy of Fallot, aortic stenosis, Marfan syndrome, aortic stenosis.

Yang Lin's predisposition to endocarditis: abnormal condition of the aortic arch in Siring in the USA (20%).

2. Causes of Loire Heart
Intravenous drug abuse, midwifery procedures, circular catheterization, dental fogging, chronic hemodialysis, labor diagnostic procedures, such as these are the most difficult circumstances.

2.3 Pathogens
Endocarditis is a common disease of the mucous membranes of the body. Bad bacteria ( S. aureus ) can attach directly to the infected body or sub-endothelial layer, and microorganisms can attach to the thrombus.

If you oppose the activity of serum bactericidal and bactericidal peptides released by platelets, if you enter a procoagulant state in the field of body tissue lysis factor and attached monocytes, in S. aureus , yariium monooth . .

Algae fibrin, tissue coagulation factors, platelet aggregation, tissue stimulating factors, multiplication of microorganisms, phytotoxicosis mangadeja.

2.4 Clinical signs of bacterial endocarditis
Symptoms of clinical bleeding appear two days after the causative precipitation. The onset is usually gradual. Bella Coman has a high satisfying ability starting from the Cold and Hot Yang Tingyi. A common manifestation of clinical disease is 5-alpha albicans endocarditis.

  1. Failure Hanging Jam. The incidence of blockage is 30-40% Spy, this is the result of the parasite Vegetation of the jungtung
  2. Bacteremia, segmental weakness - common symptoms such as fever, increased appetite, anemia, splenomegaly, metastases of distant fourth infection. Other symptoms: general weakness, nairi kibala, blindness, arthralgia pain
  3. Rexie's hypergammaglobulinemia, immune complex formation, rheumatoid factor (+), inflammation later on endocarditis, higher levels of immune complexes, this causes petechiae.
  4. Janway's defeat, namely the red spots that are not jealous. Other possible signs are splenic, kidney, and intestinal infarction, namely baldness, abdominal infarction, and signs of peritonitis. Heart disease can cause death from heart disease.
Descriptive clinical manifestations include: arthritis, glomerulonephritis, tendinitis, nausea, uremia, proteinuria, hematuria.

Subacute bacterial endocarditis clinic.

Disease course, slow grip like grip With symptoms: fatigue, anorexia, cephalafia and subfebrile condition. It can be acute in case of embolic symptoms and flatulence. The course of the disease depends on the method of taking the drug.

Tersere Disorders Bsa Kelnan Tematan Manberi. The causes of death were secondary hepatitis, swelling of the brain, and kidney failure (uremia). Endocarditis of the subacute and heart glands Group:

  1. Symptoms of generalized septicemia: Dimam tiros muteer, Mukyak also pyrexia, profuse sweating and chills, can lead to anemia.
  2. Open embolic adenoids.
  3. Heart failure in Presence: Positive noise, sometimes Balag-Uuba mauna or a new noise due to cordaktendina seber

2.5 Diagnosis of bacterial endocarditis
Bacterial endocarditis, pathogens, clinical symptoms and some laboratory tests such as blood culture to determine the cause of endocarditis by echocardiography , infective endocarditis anatomy, florametry, identification. Cardiac problems, with congenital jungtung assessment , CT diagnosis is also great for diagnosing hepatic infarct or infarction on melalloy angiography ababa dabata cymbals of titama bada kybala saket.

2.6 The diagnosis is bacterial endocarditis
Residif trama disease, sometimes very difficult to distinguish because there are niri senti. Signs of EB For example Petechia, hematuria, splenomegaly, etc. should be checked.

  1. Suffering from endocarditis
  2. empoli

2.7 Rewards
Infectious endocarditis causes cerebral embolism. Treatment: Penicillin G 4 x 3 million units/day for 4 weeks, followed by gentamicin 3 x 1 mg/coagulant/day for 2 days.

Surgery should be considered while the patient is on antibiotic treatment. On echocardiography, there is more than one major embolism larger than 10 mm.

White-pad CT scan for angiography of fungi, hematomas, and abscesses greater than 2 cm in diameter. Contact the Siargarh Office.

Congenital aneurysm-induced anticoagulation and selected anticoagulant use induce bleeding. Celine is treated with bronchodilators (theophylline, aminophylline, pentoxifylline, terbutaline) and corticosteroids (prednisolone, prednisolone, methylprednisolone).

3.1 Conclusion
Endocarditis - yangbian endocarditis is a superficial infection, which is the ventricular wall, blockage of the heart arteries, the walls of the large arteries, the septum, which is characterized by a mixture of thrombin and argin, called the yangbian virus called the yangbian virus. What happens is the general sign of the shell, strong breathing (gasping), loud voice, giving, giving, but letting go, and watery discharge from the nose.

Congestive heart failure, is an obstruction in 30-40% of cases, this is the result of parasites. Vegetation of the heart, bacteremia, jungtung physics, general symptoms such as fever, bulguri appetite, seli, metagification.

Other suspicious symptoms include: general weakness, headache, back pain, joint pain, immune reaction caused by hypergammaglobulinemia, immune complex formation, rheumatoid factor (+), endocarditis, viral infection, endocarditis, viral infection causing immune complexes, - Minibabcan tiradine hypergammaglobulinemia. The Unnerving.

Other possible signs are splenic, kidney, and intestinal infarction, namely baldness, abdominal infarction, and signs of peritonitis.

Descriptive clinical manifestations include: arthritis, glomerulonephritis, tendinitis, nausea, uremia, proteinuria, hematuria. Treatment: Gutta penicillin G units 4x3/day for 4 weeks, then followed by gentamicin 3x1 mg/coagulant/day for 2 weeks, as well as blood chemistry examination, echocardiography, and CT scan.

Postaka notebook

Indonesian Medical University. Boku Root cardiologist. Jakarta, 1996

Ramali and Ahmed Pamwanczak. T. Laksman, Hendra. Medical dictionary . Anti-ADC 2000


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